Whitney Petrosky

Research Teaching Specialist II
CABM, 213
Partridge Lab

Education

Bachelor of Science, Rutgers University/Cook College 1998

Bio

Whitney has over twenty-five year’s experience,in research starting as an undergraduate here at CABM, moving to the Cancer Institute of NJ and then back again to CABM. She is a driven professional with a unique combination of management skills and technical expertise in cancer research and neurological degenerative brain diseases.

Whitney is experienced in laboratory management, animal colony management, extensive prostate cancer and breast cancer research, in vivo imaging using multiple modalities, small animal survival surgery, dissection, histology, extensive tissue culture experience, all aspects of DNA cloning and manipulation, protein transfection and general laboratory protocols.

Whitney’s Professional Goals include continuing to grow in leadership, excel in innovating approaches to research, interact and share with team members and colleagues my knowledge and experience, and to develop and contribute to the field of cancer research, science, and health care.

Research Focus

Cancer research, neurodegenerative brain disease research, drug discovery for osteoporosis
1.
Banach-Petrosky W, Ouyang X, Gao H, et al. Vitamin D inhibits the formation of prostatic intraepithelial neoplasia in Nkx3.1;Pten mutant mice. Clinical cancer research : an official journal of the American Association for Cancer Research. 2006;12(19):5895-901.
1.
Banach-Petrosky W, Jessen W, Ouyang X, et al. Prolonged exposure to reduced levels of androgen accelerates prostate cancer progression in Nkx3.1; Pten mutant mice. Cancer research. 2007;67(19):9089-96.
1.
Ganapathy V, Ge R, Grazioli A, et al. Targeting the Transforming Growth Factor-beta pathway inhibits human basal-like breast cancer metastasis. Molecular cancer. 2010;9:122. doi:10.1186/1476-4598-9-122.
1.
Kantamneni H, Zevon M, Donzanti M, et al. Surveillance nanotechnology for multi-organ cancer metastases. Nature biomedical engineering. 2017;1:993-1003. doi:10.1038/s41551-017-0167-9.
1.
Paratala B, Chung J, Williams C, et al. RET rearrangements are actionable alterations in breast cancer. Nature communications. 2018;9(1):4821. doi:10.1038/s41467-018-07341-4.
1.
Sleat D, Maita I, Banach-Petrosky W, et al. Elevated levels of tripeptidyl peptidase 1 do not ameliorate pathogenesis in a mouse model of Alzheimer disease. Neurobiology of aging. 2022;118:106-107. doi:10.1016/j.neurobiolaging.2022.06.012.
1.
Sleat D, Banach-Petrosky W, Larrimore K, et al. A mouse mutant deficient in both neuronal ceroid lipofuscinosis-associated proteins CLN3 and TPP1. Journal of inherited metabolic disease. 2023;46(4):720-734. doi:10.1002/jimd.12619.